Acute Myocardial Infarction (MI) can be defined as condition in which, there is death or necrosis of myocardial cells. It is generally diagnosed at the end of the ambit of myocardial ischemia or acute coronary condition. Myocardial infarction occurs once myocardial ischemia oversteps a critical threshold and overcomes myocardial cellular repairing mechanism that is contrived for maintaining normal operating function and hemostasis. Ischemia at such critical threshold level for quite a longer period ends up in irreversible myocardial cell damage or in worst cases, even its death.
Most of the cases of myocardial infarction are caused due to a commotion in the vascular endothelium that is linked with unstable atherosclerotic plaques those stimulate the formation of an intracoronary thrombus that ultimately ends up in coronary artery blood flow block. If such blockage persists for longer duration (approximately 20 to 40 min) there will be irreversible myocardial cell damage and cell death. The development of atherosclerotic plaques occurs over a period of years and even to the decades. However, the reason for initial vascular lesion that leads to the development of atherosclerotic plaques is still illusive.
Generally, it is high level of cholesterols in the bloodstream that restrict the blood flow to the cardiac tissues. Excessive body fats, high cholesterol and/or lipid profiles can be few of the main culprits for myocardial infarction. The blood vessels are nothing but the hollow pipe or tube-like cylindrical structure that allows the blood to flow flawlessly. High cholesterol levels start accumulating onto the walls of blood vessels and this, in turn, results in narrowing of the blood vessels. The blood supply is reduced and over the times, it could be cut-off for a while (ischemic conditions, where there is transient loss of blood supply). When the affected organ is heart, it is ischemic heart attack.
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1 Komentar untuk "Pathway AMI"
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